Crocodile hunter.

[felixalbers]

>From somewhere in the internet:

The toxins are mostly made up of 5-nucleotidase and phosphodiesterase which are cytotoxins (they kill cells/tissue) and result in local necrosis (tissue death). This can lead to secondary bacterial infection and severe wounds may take months to heal. Stingray toxin also contains serotonin, which causes severe localized pain lasting up to 48 hours.

The toxins are also potent vasoconstrictors i.e. the proteins cause arteries and capillaries at the injection site to constrict and reduce blood flow. This property is common among venomous animals and acts to restrict blood flow to the wound so that the other toxins stick around and cause their damage without being carried away or diluted by blood flow.

However if enough of this venom gets into the circulation it can cause constriction of the coronary vessels leading to cardiac ischemia which itself can lead to arrhythmias and low blood pressure from decreased cardiac output.


Felix

De:trauma-list-bounces at trauma.org

Para:"Trauma & Critical Care mailing list" trauma-list at trauma.org

Cópia:

Data:Wed, 6 Sep 2006 21:48:11 -0000

Assunto:Re: Crocodile hunter.

> Wait for the necropsy report, or flip a coin.
> 
> Pret
> ----- Original Message -----
> From: 
> To: 
> Sent: Wednesday, September 06, 2006 10:00 PM
> Subject: Re: Crocodile hunter.
> 
> 
> > Clarification: Do the members of this list believe cardiac arrest ensued
> > secondary to cardiac tamponade from myocardial or coronary puncture? or
> do
> > members believe that venom contributed in some way to the arrest?
> >
> > DN
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